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COVID-19 Side Effects: Even after recovering from COVID-19 infection, the number of deaths due to it does not seem to be decreasing. Recently, the truth about heart attack and stroke caused by the COVID vaccine Covishield was revealed. Now a study has revealed the reason behind lung damage and death in COVID patients.
Its side effects may still be present in patients who have recovered from Covid-19 infection. Due to this, cases of deterioration in health, heart attack, stroke and lung failure and deaths due to it are continuously increasing.
In a study published in the journal Nature Communications The reason behind lung damage in COVID patients has been revealed. According to this, lung cells of patients infected with COVID-19 start dying rapidly. This can lead to life-threatening conditions like inflammation in the lungs and acute respiratory disorders. Researchers have also reported that new methods can be developed to treat lung diseases caused by COVID-19 by preventing ferroptosis.
What is Ferroptosis?
Cell death is a natural process in which cells stop working. During cell death, the molecules inside the cell break down. This process occurs naturally in humans with illness or aging. But ferroptosis is an abnormal form of cell death in which the outer fat layers of cells break down and they begin to die and the lungs become weak.
Sample taken from dead body
Researchers at Columbia University in the US This study has been done. They analyzed the tissues of patients who died due to respiratory failure caused by COVID-19. The tissues of hamsters were also analyzed in the study.
Relation of COVID-19 and Ferroptosis
The results of the study show that the amount of ferroptosis in the lungs of patients severely affected by COVID-19 is much higher than normal. This is the reason why their lungs get damaged so much.
A cure for COVID-19 may be found
Researchers hope that the development of drugs that inhibit ferroptosis could greatly help in the treatment of COVID-19. This study is still in its early stages and more research is needed on drugs that target ferroptosis. However, this study shows a new direction for the treatment of COVID-19.
